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Ling Yin on Muck Rack

Ling Yin

As seen in: Nature, Wiley Online Library, BMJ Open, Frontiers, MDPI, SAGE Publishing, Taylor & Francis Online, Genome Medicine, Science Magazine, Royal Society of Chemistry and
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Articles

Polymer Brush-Mediated Surface-Initiated Polycondensation Enables sp 2 C-COF Thin Films with Controlled Crystallinity and High Photocatalytic Performance

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By Lingjie He, Yuxiang Zhao, Guangen Fu, Ling Yin, Wenkai Zhao, Mengwei Chen, Jun He, Liping Wang, Tao Zhang, Wuxin Zhang
| Nature Verified
Abstract Sp2-carbon-conjugated covalent organic frameworks (sp2C-COFs) hold exceptional potential as π-conjugated semiconductors for optoelectronics and photocatalysis, yet their thin-film fabrication suffers from irreversible C = C linkages that impede crystallinity and morphology control. Herein, we report a polymer brush-mediated surface-initiated polycondensation (PB-SIP) strategy to synthesize the sp2C-COF films with controlled crystallinity and morphology.
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The “Spiked Helmet Sign” on Electrocardiogram Following Left Ventricular Assist Device Implantation: A Case Report

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By Qin Qian, Ling Yin
| Wiley Online Library Verified
1 Introduction Electrocardiographic (ECG) changes can provide crucial early clues to these complications. The “Spiked Helmet Sign” (also descriptively termed saddle-back ST elevation) is a distinctive ECG pattern involving diffuse, upwardly concave ST-segment elevation across multiple leads.
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Loss of human SMC5/6 induces spindle multipolarity and genome stress exacerbated by BLM helicase deficiency

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By Jialing Fu, Chao Jin, Huimin Zhang, Tiantian Ma, Dandan Zhu, Ling Yin, Victoria Murphey, Xiaolan Zhao, Junjie Chen
| Nature Verified
Abstract Mutations of the human SMC5/6 genome maintenance complex can lead to several genome instability syndromes and are associated with tumorigenesis. However, the molecular consequences of SMC5/6 deficiencies in human cells are only partly known, preventing a better understanding of disease-causing mechanisms. To address this issue, we employed the dTAG technology to induce rapid loss of SMC5 or SMC6 in a panel of human cell lines.
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