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Livia Metani

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Articles

Mitochondrial energetic failure underlies FLVCR1-related sensory neuropathy - Communications Biology

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By Francesca Bertino, Eleonora Grasso, Joanna Kopecka, Chiara Salio, Barbara Gnutti, Ram Manohar Basnet, Stefania Bellini, Luca Mignani, Boxun Zhao, Felix Kleefeld, Andreas Hentschel Verified, Francesca Magnani, Veronica Fiorito, Anna Lucia Allocco, Sara Petrillo, Francesco De Giorgio, Giorgia Ammirata, Ettore Salsano, Davide Pareyson, Maja Di Rocco, Angela Abicht, Emily A. McCourt, Rita Horvath, Heike Kölbel, Austin Larson, Andreas Roos, Timothy Yu, Dario Finazzi, Chiara Riganti, Deborah Chiabrando, Raluca Elena Abalai, Livia Metani, Emanuela Tolosano
| Nature Verified
Abstract Genetic pain loss disorders represent a heterogeneous group of rare diseases mainly characterized by defective nociception. Understanding the underlying molecular mechanism is fundamental to improve the treatment of patients affected by these rare disorders. Feline Leukemia Virus Subgroup C Receptor 1 (FLVCR1) is one of the genes previously associated with sensory neuropathy that requires further investigation.
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