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Lo Cousin

As seen in: BioRxiv
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Articles

Morphological profiling reveals neuroprotection via mitochondrial uncoupling in human dopaminergic neurons

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By Vyron Gorgogietas, Amélie Weiss, Lo Cousin, David Hoffmann
| BioRxiv
Abstract Parkinson's disease (PD) involves multiple pathological processes in midbrain dopaminergic (mDA) neurons, including protein degradation defects, vesicular trafficking disruption, endolysosomal dysfunction, mitochondrial issues, and oxidative stress. Current PD models often lack complexity and focus on single phenotypes.
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Morphological profiling in human dopaminergic neurons identifies mitochondrial uncoupling as a neuroprotective effect

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By Amélie Weiss, David Hoffmann, Vyron Gorgogietas, Lo Cousin
| BioRxiv
Abstract Multiple pathological cell biological processes in midbrain dopaminergic (mDA) neurons contribute to Parkinson's disease (PD). Described disease mechanisms converge upon defects in protein degradation, disruption of vesicular trafficking, endolysosomal function, mitochondrial dysfunction and oxidative stress.
Open in Who Shared Issue with article?
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