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Numerous human pathogens express AOX, the activity of which facilitates a number of vital functions in the pathogen adaptation processes, which in turn can enhance their pathogenicity. One example is in Trypanosoma brucei (a causative agent of trypanosomiasis), where AOX plays a crucial role in adapting the metabolism to facilitate its life-cycle either in the tsetse fly (genus Glossina) or within the human bloodstream.
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