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Matthias Bartneck on Muck Rack

Matthias Bartneck

As seen in: Nature, Journal of Intensive Care
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Articles

Expression of Cyclin E1 in hepatic stellate cells is critical for the induction and progression of liver fibrosis and hepatocellular carcinoma in mice | Cell Death & Disease

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By Tobias Otto, Ralf Weiskirchen, Matthias Bartneck, Christian Trautwein, Anna Verwaayen, Christian Penners
| Nature Verified
Abstract Hepatocellular carcinoma (HCC) is one of the most severe malignancies with increasing incidence and limited treatment options. Typically, HCC develops during a multistep process involving chronic liver inflammation and liver fibrosis. The latter is characterized by the accumulation of extracellular matrix produced by Hepatic Stellate Cells (HSCs).
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Correction to: Neutrophils are a main source of circulating suPAR predicting outcome in critical illness

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By Hendrik Gussen, Philipp Hohlstein, Matthias Bartneck, Klaudia Theresa Warzecha
| Journal of Intensive Care Verified
Hendrik Gussen1, Philipp Hohlstein1, Matthias Bartneck1, Klaudia Theresa Warzecha1, Lukas Buendgens1, Tom Luedde1, Christian Trautwein1, Alexander Koch1 & Frank Tacke1,2 Journal of Intensive Carevolume 7, Article number: 40 (2019) | Download Citation The original article was published in Journal of Intensive Care 2019 7:26 Correction to: J Intensive Care (2019) 7:26 https://10.1186/s40560-019-0381-5 In the original publication of this article [1], the content of Authors’ contributions needs...
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Neutrophils are a main source of circulating suPAR predicting outcome in critical illness

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By Hendrik Gussen, Philipp Hohlstein, Matthias Bartneck, Klaudia Theresa Warzecha
| Journal of Intensive Care Verified
Journal of Intensive Care20197:26 https://doi.org/10.1186/s40560-019-0381-5 ©  The Author(s). 2019 Received: 16 February 2019 Accepted: 12 April 2019 Published: 27 April 2019 Abstract Background Circulating levels of soluble urokinase plasminogen activation receptor (suPAR) have been proposed as a prognostic biomarker in patients with critical illness and sepsis. However, the origin of suPAR in sepsis has remained obscure.
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