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Metabolic Neuroscience

As seen in: Nature, Wiley Online Library, Oxford University Press (OUP), Science Magazine, JNeurosci, Diabetes Magazine, Brain Stimulation, Autonomic Neuroscience, JHEP Reports, The European Heart Journal Podcast
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Articles

Non-invasive vagus nerve stimulation and exercise capacity in healthy volunteers: a randomized trial

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By Gutierrez del Arroyo, Tom E F, Metabolic Neuroscience, Amour B U
| The European Heart Journal Podcast
Maintaining physical activity is essential for every aspect of cardiovascular, emotional, and cognitive health.1 Higher exercise capacity is strongly associated with a reduced risk of cardiovascular disease, type 2 diabetes, malignancy, neurodegenerative disease, and premature death.2 Regular exercise is required to maintain and improve cardiorespiratory fitness.3 Programmed moderate-to-vigorous physical activity has been demonstrated to be the most effective and time-efficient protective...
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On the regulation of arterial blood pressure by an intracranial baroreceptor mechanism

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By Metabolic Neuroscience, Fiona D. McBryde, Alla Korsak, Philippa Wittenberg
| Wiley Online Library Verified
Introduction Systemic arterial blood pressure (ABP), heart rate (HR) and cardiac contractility are controlled by neural circuits in the brainstem, which continually fine-tune sympathetic vasomotor and cardiac sympathetic and parasympathetic nerve activities in accord with the currently prevailing physiological and behavioural needs. This intricate regulation depends on afferent information received by the brainstem autonomic control circuits from various sources.
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On the mechanisms of brain blood flow regulation during hypoxia

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By Alexander Mascarenhas, Metabolic Neuroscience, Alice Braga, Sara Maria Majernikova
| Wiley Online Library Verified
The human brain consumes ∼20% of inspired oxygen at rest and up to 30% under conditions of increased mental and/or physical demand (Attwell & Laughlin, 2001; Howarth et al., 2012). The exceptionally high energy requirements of the brain make it susceptible to damage in conditions of insufficient supply of oxygen. If brain blood supply were to suddenly cease, the cerebral oxygen content (∼0.03 mm) would be sufficient to maintain neuronal activity for only a few seconds (Bailey, 2019; Buxton, 2010).
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