Mingce Zhang on Muck Rack

Mingce Zhang

As seen in: MDPI, medRxiv, The Journal of Immunology
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Articles

Role of DOCK8 in Hyper-inflammatory Syndromes

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By Mingce Zhang, Niansheng Chu, Scott Gordon, Remy R Cron
| medRxiv
R.Q.C. and E.M.B. have received clinical trial grant support and consulting fees from Sobi (manufacturer of anakinra). R.Q.C. and S.W.C. received speaking fees from Sobi and Practicepoint, CME. There are no other relevant financial disclosures. The work was supported by grants from the Histiocytosis Association (to R.Q.C.), the Kaul Pediatric Research Institute (to R.Q.C.), NIH R01-HD-098428 (to S.W.C.), and NIH R01-AI-121250-A1 (to E.M.B.).
Open in Who Shared View Duplicates (53) Issue with article?

DOCK2 Mutation and Recurrent Hemophagocytic Lymphohistiocytosis

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By Daniel Reiff, Mingce Zhang, Randy Q Cron
| MDPI
Abstract : Hemophagocytic lymphohistiocytosis (HLH) is a syndrome resulting from uncontrolled hyper-inflammation, excessive immune system activation, and elevated levels of inflammatory cytokines. HLH can be caused by the inability to downregulate activated macrophages by natural killer (NK) and CD8 cytotoxic T cells through a process reliant on perforin and granzyme B to initiate apoptosis.
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A heterozygous RAB27A mutation causes delayed cytolytic granule polarization and hemophagocytic lymphohistiocytosis in 2 unrelated teenagers

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By Randy Q Cron, Mingce Zhang, Claudia Bracaglia, Giusi Prencipe, Christina J Bemrich-Stolz
| The Journal of Immunology
Frequently fatal, primary hemophagocytic lymphohistiocytosis (HLH) occurs in infancy resulting from homozygous mutations in natural killer (NK) and CD8 T cell cytolytic pathway genes. Secondary HLH presents after infancy and may be associated with heterozygous mutations in HLH genes. We report 2 unrelated teenagers with HLH and an identical heterozygous RAB27A mutation (259 G>C).
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