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Miyako Rogers on Muck Rack

Miyako Rogers

Manchester
As seen in: SAGE Publishing, BioRxiv, Front Line Genomics
Science Writer ¦ MSci Biomedical Sciences with Honours ¦ Special interest in Multi-Omics, Machine Learning, Neuroscience, Immunology

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Articles

Selective deletion of interleukin-1 alpha in microglia does not modify acute outcome but may regulate neurorepair processes after experimental ischemic stroke - Eloïse Lemarchand, Alba Grayston, Raymond Wong, Miyako Rogers, Blake Ouvrier, Benjamin Llewell

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By Eloïse Lemarchand, Alba Grayston, Raymond Wong, Miyako Rogers
| SAGE Publishing
Cite article If you have citation software installed, you can download article citation data to the citation manager of your choice Select your citation manager software: Share options Share this article Share with email Email Article Link Share on social media Share access to this article Sharing links are not relevant where the article is open access and not available if you do not have a subscription. For more information view the Sage Journals article sharing page.
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Selective deletion of interleukin-1 alpha in microglia does not modify acute outcome but regulates neurorepair processes after experimental ischemic stroke.

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By Eloïse Lemarchand, Alba Grayston, Raymond Wong, Miyako Rogers
| BioRxiv
Abstract Inflammation is a key contributor to stroke pathogenesis and exacerbates brain damage leading to poor outcome. Interleukin-1 (IL-1) is an important regulator of post-stroke inflammation, and blocking its actions is beneficial in pre-clinical stroke models and safe in the clinical setting. However, the distinct roles of the two major IL-1 receptor type 1 agonists, IL-1α and IL-1β, and the specific role of IL-1α in ischemic stroke remain largely unknown.
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Selective deletion of interleukin-1 alpha in microglia regulates neuronal activity and neurorepair processes after experimental ischemic stroke.

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By Eloïse Lemarchand, Raymond Wong, Miyako Rogers, Alba Grayston
| BioRxiv
Abstract Inflammation is a key contributor to stroke pathogenesis and drives exacerbated brain damage leading to poor outcomes in patients. Interleukin-1 (IL-1) is an important regulator of post-stroke inflammation, and blocking its actions is beneficial in pre-clinical stroke models and safe in the clinical setting.
Open in Who Shared Issue with article?
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