Moriam Akter Munni

Introduction Guillain–Barré syndrome (GBS) is an autoimmune-mediated disorder affecting the axons and myelin sheath of the peripheral nervous system (PNS) with high clinical disability.1 Presently, GBS is considered as an excellent paradigm of molecular mimicry in which lipo-oligosaccharide in the outer core structure of the infectious agents induce the formation of cross-reactive antibodies against host nerve gangliosides.2 These pathogenic antibodies result in an aberrant immune system and...