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Damage to the CNS can cause a differential spatio-temporal release of multiple factors, such as nucleotides, ATP and UTP. The latter interact with neuronal and glial nucleotide receptors. The P2Y nucleotide receptor (P2Y R) has gained prominence as a modulator of gliotic responses after CNS injury. Still, the molecular mechanisms underlying these responses in glia are not fully understood.
Damage to the CNS can cause a differential spatio-temporal release of multiple factors, such as nucleotides like ATP and UTP. The latter interact with neuronal and glial nucleotide receptors. The P2Y nucleotide receptor (P2Y R) has gained prominence as a modulator of gliotic responses after CNS injury. Still, the molecular mechanisms underlying these responses in glia are not fully understood.
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