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Oriola Gjana

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The SIRT1 activator SRT2104 exerts exercise mimetic effects and promotes Duchenne muscular dystrophy recovery - Cell Death & Disease

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By Matteo Giovarelli, Silvia Casati, Elisabetta Catalani, Annapaola Andolfo, Clarissa Braccia, Emilio Clementi, Davide Cervia, Cristiana Perrotta, Laura Lociuro, Oriola Gjana, Anne Bigot
| Nature Verified
Abstract Duchenne muscular dystrophy (DMD) is a devastating genetic disorder, whose management is still a major challenge, despite progress in genetic and pharmacological disease-modifying treatments have been made. Mitochondrial dysfunctions contribute to DMD, however, there are no effective mitochondrial therapies for DMD. SIRT1 is a NAD+-dependent deacetylase that controls several key processes and whose impairment is involved in determining mitochondrial dysfunction in DMD.
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