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Pankaj Sing Parihar on Muck Rack

Pankaj Sing Parihar

As seen in: BioRxiv
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Articles

Extended regulation interface coupled to the allosteric network and disease mutations in the PP2A-B56δ holoenzyme

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By Cheng-Guo Wu, Vijaya Kumar Balakrishnan, Pankaj Sing Parihar, Kirill Konovolov
| BioRxiv
Abstract An increasing number of mutations associated with devastating human diseases are diagnosed by whole-genome/exon sequencing. Recurrent de novo missense mutations have been discovered in B56δ (encoded by PPP2R5D), a regulatory subunit of protein phosphatase 2A (PP2A), that cause intellectual disabilities (ID), macrocephaly, Parkinsonism, and a broad range of neurological symptoms.
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Extended regulation interface coupled to the allosteric network and disease mutations in the PP2A-B56 delta holoenzyme

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By Cheng-Guo Wu, Vijaya Kumar Balakrishnan, Pankaj Sing Parihar, Kirill Konovolov
| BioRxiv
Thank you for your interest in spreading the word about bioRxiv. NOTE: Your email address is requested solely to identify you as the sender of this article. Your Email * Your Name * Send To * Enter multiple addresses on separate lines or separate them with commas. Message Subject (Your Name) has forwarded a page to you from bioRxiv Message Body (Your Name) thought you would like to see this page from the bioRxiv website.
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