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Wild-type p53 overexpression in NPM1-mutated acute myeloid leukemia: potential implications for disease biology and therapy response

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By Siarhei Dzedzik, Paul D. Barone, Ashley S. Kleinman
| haematologica.org
In the latest World Health Organization (WHO) and International Consensus Classification (ICC) diagnostic schema for hematolymphoid neoplasms acute myeloid leukemias (AML) are now largely genetically-defined.1,2 At seemingly opposite ends of the clinicopathologic and genomic spectra lie AML with mutated NPM1 (NPM1-AML) and AML (and precursor states) harboring TP53 abnormalities (TP53-AML).
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