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Paul James

Manchester
As seen in: High Times, Medium, Nature, Wiley Online Library, Oxford Academic, Frontiers, MDPI, ZDNet, Biomed Central (BMC), Florence Morning News, People Matters and
MD darc media, publisher of arc and darc, the international magazines for lighting in architecture. Organiser of darc awards, darc sessions and LiGHT.
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Articles

Stearoyl-CoA desaturase 1-mediated lipid metabolic reprogramming in granulosa cells contributes to follicular dysfunction in non-overweight polycystic ovary syndrome patients

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By Zhejiang Gongshang, Paul James
| Oxford Academic Verified
In women of reproductive age, polycystic ovary syndrome (PCOS) is a widespread endocrine condition that is primarily connected to disrupted lipid metabolism. However, the cell-specific lipidomic signatures and their regulatory mechanisms in ovarian granulosa cells (GCs) of PCOS women remain elusive, particularly across clinical subgroups. We conducted quantitative lipidomics to profile luteinized GC lipid metabolism in PCOS subgroups.
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Navigating men’s health: Questions to ask your provider

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By Paul James, Dnp HopeHealth
| Florence Morning News Verified
Men’s health has received increased attention in recent years. Many men come to my office to establish care and say things like, “I haven’t seen a doctor in years,” or, my personal favorite, “I’m only here because my wife told me to come.” kAmp=E9@F89 E96C6 2C6 2=H2JD 6I46AE:@?D[ x 92G6 7@F?5 E92E >6? 2C6 @7E6? >@C6 =:<6=J E92? H@>6? E@ 56=2J E96:C @H? D6=7\42C6]k^Am kAm}2E:@?2= 52E2 DFAA@CED E9:D A2EE6C?[ D9@H:?8 E92E H@>6?
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Cardiorespiratory fitness, genetic susceptibility, and the risk of chronic obstructive pulmonary disease: findings from observational and two-sample bidirectional Mendelian randomisation analyses

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By Paul James, Shiu Lun Au, LKS Faculty
| link.springer.com
Background Cardiorespiratory fitness (CRF) is a common risk factor for cardiometabolic diseases, but the causal relation of CRF with chronic obstructive pulmonary disease (COPD) and its interplay with genetic risk remain unknown. We integrated genetic susceptibility and causal inference methods to evaluate the protective roles of CRF in COPD development. Methods We included 68,288 White British individuals from the UK Biobank (aged 40–79 years) without prevalent COPD at the baseline (2006–13).
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