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Pengqing Nie

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Articles

The Compromised Fanconi Anemia Pathway in Prelamin A‐Expressing Cells Contributes to Replication Stress‐Induced Genomic Instability

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By Cheng Zhang, Fengyi Wu, Shi Chen, Pengqing Nie
| Wiley Online Library Verified
1 Introduction The alternative splicing products of the lamin A/C (LMNA) gene, lamin A and lamin C, are major components of the nuclear skeleton.[1] Mature lamin A is produced by posttranslational processing of the initial translation product prelamin A.[2] Mutations in the LMNA gene are closely related to Hutchinson–Gilford progeria syndrome (HGPS).
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