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Pontian Adogamhe

As seen in: BioRxiv
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Articles

The mitochondrial unfolded protein response and metabolic reprogramming promote PASMC proliferation in response to sphingosine kinase-1/sphingosine-1-phosphate signaling

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By Angelia Lockett, Aaron Snow, Justin R. Sysol, Pontian Adogamhe
| BioRxiv
Abstract Proliferation and vasoconstriction of the intimal smooth muscle layer of the pulmonary artery are pathogenic characteristics of pulmonary arterial hypertension (PAH). Altered mitochondrial function, i.e. glycolysis, ROS generation and fission, are known potentiators of vascular remodeling.
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