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Punchappady Devasya on Muck Rack

Punchappady Devasya

As seen in: Oxford University Press (OUP), Frontiers
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Articles

Dysbiosis of the oral microbiota composition is associated with oral squamous cell carcinoma and the impact of radiotherapy: A pilot study

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By Maxillofacial Diseases, Punchappady Devasya, Mangesh Vasant, Ananthapadmanabha Bhagwath
| Oxford University Press (OUP)
Radiotherapy can potentially influence the diversity and composition of the oral microbiome. We performed a study comparing the composition of oral microbiota in patients with oral squamous cell carcinoma (OSCC) before radiotherapy (n = 6), at three months (n = 6), and six months (n = 6) post-radiotherapy, and controls (n = 6). We profiled the oral microbiome by 16S rRNA gene sequencing using Illumina MiSeq.
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Serum autoantibody profiling of oral squamous cell carcinoma patients reveals NUBP2 as a potential diagnostic marker

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By Punchappady Devasya, Shanaya Patel, Devasia Puthenpurackal, Riaz Abdulla
| Frontiers
1 Introduction Cancers of the lip and the oral cavity constitute the 16th most common neoplasms in the world, with 377,713 new cases (Age-standardized incidence: 4.1) and 177,757 mortalities (Age-standardized incidence: 1.9) estimated in 2020 (1). The highest incidences of these cancers have been reported in Papua New Guinea, Pakistan, Latvia, India, and Bangladesh (2). Oral squamous cell carcinoma (OSCC) cases constitute 90% of lip and oral cavity cancers.
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Pleotropic potential of quorum sensing mediated N-acyl homoserine lactones (AHLs) at the LasR and RhlR receptors of Pseudomonas aeruginosa - Structural Chemistry

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By Devasahayam Arokia Balaya, Punchappady Devasya
| link.springer.com
Abstract Pseudomonas aeruginosa is an opportunistic pathogen, having complicated quorum sensing (QS) system utilizing multiple signals and receptors to coordinate virulence and pathogenicity. N-acyl-homoserine-lactones (AHLs) are the most common autoinducers responsible for regulation of QS-mediated virulence gene expression. There are four QS systems in P. aeruginosa among which the LasI/R and RhlI/R systems are regulated by 3-oxo-C12-HSL and C4-HSL respectively.
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