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Qinkang Lu on Muck Rack

Qinkang Lu

As seen in: Nature, Frontiers
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Articles

Aqueous humour extracellular vesicle membrane protein profiling reveals pathological features of refractory macular edema

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By Yufei Wu, Yujie Wang, Xiaodong Xie, Weina Ren, Yitian Zhao, Qinkang Lu, Shuanghua Xin, Zai-Long Chi
| Frontiers
Abstract Background: Both diabetic macular edema (DME) and retinal vein occlusion-related macular edema (RVO-ME) can become refractory to anti-vascular endothelial growth factor (anti-VEGF) therapy, but the underlying mechanisms are unclear. Molecular discrimination of refractory disease could guide personalized treatment. This study examined whether aqueous humor-derived extracellular vesicle (EV) membrane proteins can characterize refractoriness and reveal etiology-specific pathways.
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The broad-spectrum deubiquitinating enzyme inhibitor PR-619 protects retinal ganglion cell and augments parkin-mediated mitophagy in experimental glaucoma | Scientific Reports

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By Qinkang Lu
| Nature Verified
Abstract Glaucoma is a leading cause of irreversible visual impairment worldwide, characterized by the progressive death of retinal ganglion cells (RGCs). Deubiquitinating enzyme (DUB) inhibitors have shown promise as pharmacological interventions for neurodegenerative disorders. Our study focuses on the pan-DUB inhibitor PR-619 and its potential neuroprotective effects on RGCs through modulation of parkin-mediated mitophagy in experimental glaucoma models.
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