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Qiuying Li

As seen in: Nature, Wiley Online Library, British Pharmacological Society, PLOS ONE, MDPI, ACS Publications, Flipboard, Royal Society of Chemistry, Canadian Journal of Infectious Diseases & Medical Microbiology, Dove Medical Press and
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Articles

Potent and Sustained Lowering of Serum Uric Acid by YJH-012-D, a GalNAc-Conjugated siRNA Targeting Xanthine Dehydrogenase

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By Qiuying Li, Chenguang Zhang, Tingke Tang
| British Pharmacological Society
1 Introduction Hyperuricemia (HUA) is a chronic metabolic disorder characterized by abnormally elevated serum uric acid levels. Its global prevalence is rising, particularly in China, in parallel with population aging and changes in lifestyle [1]. A nationwide survey spanning 13 provinces reported a prevalence of 6.4% among Chinese adults [2].
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HMGA2 identified via m6A-senescence multi-omics in laryngeal squamous cell carcinoma

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By Meng Jin, Lingmei Qu, Zhaonan Xu, Shuang Teng, Shuo Liu, Qiuying Li, Fengshuo Zhang, Qing Hao, Peng Wang, Rui Zhao, Bingrui Yan, Yanan Sun
| Nature Verified
Abstract N6-methyladenosine (m6A) modification and cellular senescence have been individually implicated in tumor biology, but the landscape of m6A-related senescence genes in laryngeal squamous cell carcinoma (LSCC) remains underexplored. In this study, we conducted an integrated multi-omics analysis combining bulk and single-cell transcriptomics, somatic mutation and copy number variation data, and proteomics to explore the clinical and molecular impacts of m6A-related senescence.
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Btg2 inhibits Fmo1 UFMylation thus exacerbating ferroptosis and apoptosis in hepatic ischemia-reperfusion injury

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By Dadi Peng, Yihua Wang, Dengliang Lei, Kezhen Zong, Shanshan Li, Yunhai Luo, Qiuying Li, Liqing Jiang, Weifeng Huang, Huarong Yu, Jiayin Yang, Zuotian Huang, Zhongjun Wu, Yulin Ou
| Nature Verified
Abstract Hepatic ischemia‒reperfusion injury (HIRI) represents a frequently occurring pathological condition during liver surgery, yet the mechanisms governing HIRI remain inadequately comprehended. Here, we investigate the role of B-cell translocation gene 2 (Btg2) in HIRI. Btg2 is upregulated following HIRI. A 70% HIRI model using hepatocyte-specific Btg2 transgenic and systemic Btg2 knockout mice reveals that Btg2 deteriorates hepatic inflammation and apoptosis.
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