Quentin M Anstee on Muck Rack

Quentin M Anstee

Newcastle upon Tyne
As seen in: Color Research & Application, Nature, Wiley Online Library, Frontline Gastroenterology, Gut Journal, MDPI, Cell Metabolism, BJGP (British Journal of General Practice), Gastroenterology, Journal Of Hepatology EU and
Professor of Experimental Hepatology & Consultant Hepatologist: NAFLD/MASLD Treatment | Biomarkers | Pathogenesis | Genetics
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Articles

Food Insecurity Is Highly Prevalent Amongst Patients With Chronic Liver Disease in Northern England: A Cross‐Sectional Analysis

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By Rachel Howarth, Stuart McPherson, Quentin M Anstee, Thomas Crame
| Wiley Online Library Verified
1 Introduction Liver disease is the third leading cause of death in the United Kingdom (UK), with mortality rates having increased by 400% since 1970 [1]. This rising burden parallels the increase in excess alcohol consumption, obesity, and viral hepatitis [1]. Liver disorders disproportionately affect the most economically disadvantaged, exacerbating health inequalities [2].
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Clinical trial design, biomarkers and end points in metabolic and alcohol-related liver disease - Nature Reviews Gastroenterology & Hepatology

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By Luis Diaz, Maja Thiele, Alexandre Louvet, Brian Lee, Veeral Ajmera, Federica Tavaglione, Cynthia Hsu, Ramon Bataller, Mack C. Mitchell, Mary E. Rinella, Shiv Sarin, Vijay Shah, Meena B. Bansal, Lorenzo Leggio, Patrick S. Kamath, Aleksander Krag, Arun J Sanyal, Juan Pablo Arab, Quentin M Anstee
| Nature Verified
Abstract Metabolic and alcohol-related liver disease (MetALD) is a newly defined entity within the spectrum of steatotic liver disease, characterized by the interplay of cardiometabolic risk factors and alcohol consumption. The evolving epidemiology and complex pathophysiology of MetALD present unique challenges and opportunities for clinical trial design.
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Modulation of metabolic, inflammatory and fibrotic pathways by semaglutide in metabolic dysfunction-associated steatohepatitis - Nature Medicine

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By Jenny M. Norlin, Mette Kjær, Kasper Almholt, Lise L. Gluud, Gianluca Mazzoni, Vlad Ratziu, Vincent Wong, Quentin M Anstee, Lea M. Harder
| Nature Verified
Abstract Metabolic dysfunction-associated steatohepatitis (MASH) is a chronic liver disease strongly associated with cardiometabolic risk factors. Semaglutide, a glucagon-like peptide-1 receptor agonist, improves liver histology in MASH, but the underlying signals and pathways driving semaglutide-induced MASH resolution are not well understood.
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