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Requisite Role of Kv1.5 Channels in Coronary Metabolic Dilation

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By Nadeem Khan Verified, Raffi Bardakjian, Christopher Kolz, Molly Enrick, Tatevik Hakobyan, John Kmetz, Ian Bratz, Jordan Luli, Masaki Nagane, Huagang Hou, Periannan Kuppusamy, Jacqueline Graham, Danielle Janota, Moses O. Oyewumi, Suzanna Logan Verified, Jonathan R. Lindner
| circres.ahajournals.org
Methods and Results: In mice (wild-type [WT], Kv1.5 null [Kv1.5−/−], and Kv1.5−/− and WT with inducible, smooth muscle–specific expression of Kv1.5 channels), we measured mean arterial pressure, myocardial blood flow, myocardial tissue oxygen tension, and ejection fraction before and after inducing cardiac stress with norepinephrine. Cardiac work was estimated as the product of mean arterial pressure and heart rate.
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