Rakshya Shrestha

1 Introduction Hemangioma is a true benign neoplasm of endothelial origin. It is frequently encountered in soft tissues, presenting with swift postnatal expansion and slow spontaneous regression [1, 2]. Central hemangioma, also known as intraosseous hemangioma, occurs more frequently in the vertebral column and skull bone [3-5]. It is relatively uncommon in the mandible and maxilla, comprising only 0.5%–1% of all intraosseous tumors [4-6].

Abstract RNA virus induced excessive inflammation and impaired antiviral interferon (IFN-I) responses are associated with severe disease. This innate immune response, also referred to as dysregulated immunity, is caused by viral single-stranded RNA (ssRNA) and double-stranded-RNA (dsRNA) mediated exuberant inflammation and viral protein-induced IFN antagonism. However, key host factors and the underlying mechanism driving viral RNA-mediated dysregulated immunity are poorly defined.