Renil S. Titus on Muck Rack

Renil S. Titus

As seen in: Nature, BJU International Journal
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Articles

Comparative analysis of genitourinary toxicity following bladder outlet obstruction procedures in patients before versus after radiotherapy for localized prostate cancer: a retrospective cohort study using the TriNetX database Original

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By Renil S. Titus, Johnny S Su, Carlos Riveros, Annie Chen, Hemendra N. Shah, Raj Satkunasivam, Olamide Omidele, Ricardo Gonzalez, Ricardo González
| Nature Verified
Abstract Prior studies have reported genitourinary toxicity associated with transurethral resection of the prostate after radiation therapy (XRT) for localized prostate cancer (PCa). However, treatment options for benign prostatic hyperplasia have expanded exponentially within the last decade.
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Chemotherapy-induced activation of caspase-1 and IL-1α release by cancer cells remotely skews myelopoiesis to drive pro tumorigenic systemic neutrophil-dominant inflammation

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By Stephen Wong, Hongbo Gao, Haley Garcia, Sophie Porter, Mustafa Karabicici, Xen Ping Hoi, Crystal Shin, Lisa Bouchier-Hayes, Dimitrios Korentzelos, Chen Chen, Michael Brooks, Zheng Yin, Renil S. Titus, Lan Zhou, Yinan Gong, Yulin Li, Ziad M. El-Zaatari, Dharam Kaushik, Raj Satkunasivam, Fotis Nikolos, Keith Chan, Kazukuni Hayashi, Ethan J. Subel, Yung Hsing Huang, Mark D. Alonzo, Armine Kasabyan, Efrosini Tsouko
| Nature Verified
Abstract While chemotherapy-induced tumor cell death is known to modulate the local immune landscape, its systemic impact on distant bone marrow—a site essential for immune cell maturation—remains underexplored. Here, we show that gemcitabine chemotherapy induces inflammatory caspase-1-dependent pyroptosis in epithelial cancer cells (epiCaspase-1). Despite its inflammatory nature, epiCaspase-1-mediated cell death is non-immunogenic.
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Addition of immune checkpoint inhibitors to intravesical BCG for high-risk BCG-naïve non-muscle invasive bladder cancer: Systematic review and meta-analysis

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By Renil S. Titus, Carlos Riveros, Eusebio Luna Velasquez
| BJU International Journal Verified
1 INTRODUCTION Bladder cancer is the ninth most prevalent cancer, and approximately 75% of cases are non-muscle invasive (NMIBC) [1]. Approximately half are considered high-risk and treated by transurethral resection and intravesical induction and maintenance-BCG (BCG-I + M) [2]. Although it is associated with a high response rate, 30–40% of patients fail treatment, requiring other intravesical therapies or escalation of therapy for progression [3].
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