Russel H. Dinh

Address correspondence to Avni P. Finn, MD, MBA, Vanderbilt Eye Institute, 2311 Pierce Avenue, Nashville, TN 37232; email: avni.finn@vumc.org . From the Department of Ophthalmology, Vanderbilt University Medical Center, Nashville, Tennessee. Disclosure: APF is a consultant for Genentech; and participates on advisory boards for Allergan, Apellis, Eyepoint, Genentech, and Iveric Bio. The remaining authors have disclosed no potential conflicts of interest, financial or otherwise.

Since corticosteroids were discovered in the 1950s, they have become and remain the mainstay of treatment for uveitis.1 Although the full mechanism of corticosteroids is not understood, it is believed that the basis for the anti-inflammatory and immunosuppressive effects of corticosteroids in the eye is the activation of the glucocorticoid receptor.

Report Acute macular neuroretinopathy (AMN) is a rare, poorly understood retinal disease that most commonly occurs in young, healthy women. The most commonly proposed pathogenesis is ischemia of the deep capillary plexus, and it is often associated with a preceding respiratory or influenza-like illness. 1 Bhavsar K.V. Lin S. Rahimy E. et al. Acute macular neuroretinopathy: A comprehensive review of the literature.