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Antiphospholipid syndrome (APS) is an autoimmune disorder characterized by the presence of antiphospholipid antibodies (aPLs) that interact with phospholipids and phospholipid-binding proteins on the cell surface, leading to the activation of endothelium, platelets, and leukocytes [1]. Reported prevalence rates of APS suggest approximately 50 cases per 100,000 individuals, with published estimates of annual incidence range from 2 to 80 cases per 100,000 [2,3].
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