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Sabrina Baroni

As seen in: Nature, Epidemiology and Infection
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Articles

Th17 cells require the DNA repair sensor xeroderma pigmentosum complementation Group C to control oxidative DNA damage in a murine model

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By Jefferson Antônio Leite, Luisa Menezes-Silva, Patrick Da Silva, Samuel Oliveira, Magaiver Andrade-Silva, Camila Ferreira, Victor Yuji Yariwake, Marcella Cipelli, Talita Gonçalves, Sabrina Baroni, Howard L. Weiner, Thomas Hofmann, Stefanie Scheu, Leandro Machado Colli, Ari Waisman, Natalia Notarberardino Bos, Hedden Ranfley, Ilaria Palagi, Beatriz Leocata, Nadja Cristhina de Souza-Pinto, Sandra Marcia Muxel
| Nature Verified
Abstract T helper 17 cells play essential roles in mucosal immunity and autoimmunity, yet the mechanisms that protect these cells from oxidative DNA damage remain poorly defined. Here we show, in a murine model, that the nucleotide excision repair sensor Xeroderma Pigmentosum Complementation Group C preserves genomic stability and metabolic fitness during T helper 17 cell differentiation.
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Genomic epidemiology of SARS-CoV-2 in large university hospital cohort: the UnCoVER-Brazil project | Epidemiology & Infection | Cambridge Core

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By Sarah Slack, Francisco Barbosa-Júnior, Guilherme Silveira Castro, Sabrina Baroni
| Epidemiology and Infection Verified
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the virus that causes coronavirus disease (COVID-19), remains a global threat despite massive diagnostic testing, isolation, therapies, and vaccines. Emerging SARS-CoV-2 variants of concern (VOCs) continue to challenge these measures with increased transmissibility or virulence, escape of host antibody neutralization, and decreased efficacy of detection, therapeutics, and vaccination [Reference Aleem, Akbar Samad and Slenker1].
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