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Songjia Ni

As seen in: Nature, Arthritis Research & Therapy
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Articles

CRYAB_K92 lactylation drives hypertrophy of the ligamentum flavum via an S100A16/RAGE-mediated glycolysis-fibrosis positive feedback loop

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By Jianjun Li, Cheng Yu, Junjie Cheng, Yanlin Cao, Tao Lin, Songjia Ni, Konghe Hu, Yang Duan
| Nature Verified
Abstract Lumbar spinal stenosis (LSS) caused by hypertrophy of the ligamentum flavum (HLF) is a major cause of lower limb dysfunction; however, its underlying pathogenesis remains unclear. Here we show that metabolic reprogramming and epigenetic modifications synergistically drive HLF progression through a glycolysis-lactylation-fibrosis axis.
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Integrative analysis of genome-wide DNA methylation and single-nucleotide polymorphism identified ACSM5 as a suppressor of lumbar ligamentum flavum hypertrophy | Arthritis Research & Therapy | Full Text

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By Zhong Chen, Yang Duan, Yanlin Cao, Yenan Zhan, Sujun Qiu, Kaiqin Gong, Songjia Ni
| Arthritis Research & Therapy Verified
Abstract Background Hypertrophy of ligamentum flavum (HLF) is a common lumbar degeneration disease (LDD) with typical symptoms of low back pain and limb numbness owing to an abnormal pressure on spinal nerves. Previous studies revealed HLF might be caused by fibrosis, inflammatory, and other bio-pathways. However, a global analysis of HLF is needed severely.
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