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Abstract Gammaherpesviruses (GHV) are DNA tumor viruses that establish lifelong latent infections in lymphocytes. For viruses such as Epstein-Barr virus (EBV) and murine gammaherpesvirus 68 (MHV68), this is accomplished through a viral gene-expression program that promotes cellular proliferation and differentiation, especially of germinal center (GC) B cells. Intrinsic host mechanisms that control virus-driven cellular expansion are incompletely defined.
Highlights • Anti-tumor phenotype requires reprogramming of inflammatory monocytes/macrophages • Myeloid cell functional reprogramming is dependent on IFN-γ produced by CD4T cells • TAM reprogrammed transcriptome correlates with better prognosis in human cancer Summary Regulatory T (Treg) cell infiltration of solid tumors often correlates with poor prognosis, but their tumor-suppressive function lacks mechanistic understanding.
Speaking as a retiree who’s spent considerable time floating through Labyrinth Canyon on the Green River, I’m perplexed that Rep. John Curtis and Sen. Orrin Hatch are taking such an illogical path toward protections that this Utah gem deserves. In our busy lives, quiet recreation is hard to come by, so when I find time I head straight to Labyrinth to spend time with the grandkids, doing something I’m still capable of and that’s spiritually refreshing.
In 1787, James Madison conceived of a scientific method to distribute political power in the United States. He proposed a decennial census of population to ensure that the membership of the House of Representatives would reflect the pattern of growth of the fledging nation. From that first census emerged a whole system of social and economic measurement serving the country.
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