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Talyta C. Grippe on Muck Rack

Talyta C. Grippe

As seen in: Wiley Online Library, Frontiers, Science Magazine, BioRxiv
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Articles

Transcranial ultrasound stimulation of motor networks in Parkinson’s disease informed by local field potential dynamics

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By Olha Melnyk, Rowena Jones, Leslie Wagner, CAN SARICA, Ghazaleh Darmani, Hamidreza Ramezanpour, Marcus Neil Callister, Brendan Santyr, Talyta C. Grippe, Regina Annirood, Nasem Raies, Jean-François Nankoo, Nardin Samuel, Artur Vetkas, Anton Fomenko, Jeffrey D. Schall, Mojgan Hodaie, Suneil K. Kalia, Agessandro Abrahao, Alfonso Fasano, Samuel Pichardo, Robert Chen, Andres M. Lozano, Andrés Lozano
| Science Magazine Verified
Editor’s summary Abstract Supplementary Materials REFERENCES AND NOTES Information & Authors Metrics & Citations Check Access References Figures Tables Media Share Editor’s summary Transcranial ultrasound stimulation (TUS) is a noninvasive neuromodulation technique that allows targeting of superficial as well as deep brain regions and is therefore a potential treatment avenue for Parkinson’s disease (PD).
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Transcranial Ultrasound Stimulation of Internal Globus Pallidus Region and Motor Cortex in Parkinson's Disease

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By Yi-Ying Lin, Talyta C. Grippe, Nasem Raies, Carolyn A. Gunraj
| Wiley Online Library Verified
Bradykinesia, rigidity, and tremor, the cardinal motor symptoms of Parkinson's disease (PD), are attributed to basal ganglia-thalamocortical circuit dysfunction.1, 2 Although dopaminergic therapy alleviates motor symptoms in PD, many patients experience motor impairments even during the on-medication state and develop disabling motor complications.3, 4 Consequently, extensive research has been dedicated to exploring alternative therapies, with a particular focus on modulation of the basal...
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Dissecting the Phenotypic Spectrum and Complexity of Movement Disorders in 22q11.2 Deletion Syndrome

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By Talyta C. Grippe, Marcus Neil Callister, Tarig M Abkur, Emilio Q. Villanueva
| Wiley Online Library Verified
1 Introduction Over the last 30 years, 22q11.2 deletion syndrome (22q11.2DS) has been increasingly diagnosed [1]. This complex genomic syndrome presents with variably penetrant neuropsychiatric features, with movement disorders representing important late-occurring manifestations [2-4].
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