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Taoyingnan Li

As seen in: The Journal of Immunology
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Articles

Cutting Edge: NOX2 NADPH Oxidase Controls Infection by an Intracellular Bacterial Pathogen through Limiting the Type 1 IFN Response

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By Taoyingnan Li, Aleixo Muise, Darren E. Higgins, John H. Brumell
| The Journal of Immunology Verified
Open Access Jorge David Rojas Márquez, Taoyingnan Li, Adam R. R. McCluggage, Joel M. J. Tan, Aleixo Muise, Darren E. Higgins and John H. Brumell J Immunol January 15, 2021, 206 (2) 323-328; DOI: https://doi.org/10.4049/jimmunol.2000694 Key Points NOX2 deficiency promotes systemic L. monocytogenes infection in a type I IFN–dependent manner. NOX2 limits ISG expression in response to type I IFN. Abstract The NOX2 NADPH oxidase (NOX2) produces reactive oxygen species to kill phagosome-confined bacteria.
Open in Who Shared Issue with article?

Cutting Edge: NOX2 NADPH Oxidase Controls Infection by an Intracellular Bacterial Pathogen through Limiting the Type 1 IFN Response

|
By Taoyingnan Li, Aleixo Muise, Darren E. Higgins, John H. Brumell
| The Journal of Immunology Verified
Open Access Jorge David Rojas Márquez, Taoyingnan Li, Adam R. R. McCluggage, Joel M. J. Tan, Aleixo Muise, Darren E. Higgins and John H. Brumell J Immunol December 7, 2020, ji2000694; DOI: https://doi.org/10.4049/jimmunol.2000694 Key Points NOX2 deficiency promotes systemic L. monocytogenes infection in a type I IFN–dependent manner. NOX2 limits ISG expression in response to type I IFN. Abstract The NOX2 NADPH oxidase (NOX2) produces reactive oxygen species to kill phagosome-confined bacteria.
Open in Who Shared Issue with article?
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