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Theresa E. Hickey on Muck Rack

Theresa E. Hickey

As seen in: Nature, MDPI, Genome Biology
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Articles

A Deep Learning Approach for the Classification of Fibroglandular Breast Density in Histology Images of Human Breast Tissue

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By Leigh J. Hodson, Mohsen Dorraki, Theresa E. Hickey, Hanieh Heydarlou
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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The androgen receptor interacts with GATA3 to transcriptionally regulate a luminal epithelial cell phenotype in breast cancer - Genome Biology

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By Zoya Kikhtyak, Geraldine Laven-Law, Stephen Pederson, Elgene Lim, Jason Carroll, Wayne D. Tilley, Amy R. Dwyer, Theresa E. Hickey, Leila Hosseinzadeh, Caroline G. Puiu, Clive S. D’Santos
| Genome Biology Verified
The ZR-75-1 (ATCC #CRL-1500 pg, RRID: CVCL_0588), T-47D (HTB-133, RRID: CVCL_0553), and MDA-MB-453 (HTB-131, RRID: CVCL_0418) breast cancer cell lines were obtained from American Type Cell Culture Collection (ATCC; Manassas, VA, USA), and the MFM-223 cell line was obtained from the DMSZ-German Collection of Microorganisms and Cell Cultures (Braunschweig, Germany, RRID: CVCL_1408).
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The androgen receptor is a tumor suppressor in estrogen receptor-positive breast cancer

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By Daniel L Roden, Theresa E. Hickey, Geraldine Laven-Law, Heloisa H. Milioli
| Nature Verified
Abstract The role of the androgen receptor (AR) in estrogen receptor (ER)-α-positive breast cancer is controversial, constraining implementation of AR-directed therapies. Using a diverse, clinically relevant panel of cell-line and patient-derived models, we demonstrate that AR activation, not suppression, exerts potent antitumor activity in multiple disease contexts, including resistance to standard-of-care ER and CDK4/6 inhibitors.
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