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Tim Meyring

As seen in: BioRxiv, eLife
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Articles

DNA damage signaling in Drosophila macrophages modulates systemic cytokine levels in response to oxidative stress

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By Fabian Hersperger, Tim Meyring, Pia Weber, Chintan Chhatbar
| eLife
eLife assessment This study elucidates the role of a specific hemocyte subpopulation in oxidative damage response by establishing connections between DNA damage response and the JNK-JAK/STAT axis to regulate energy metabolism. The identification of this distinct hemocyte subpopulation through single-cell RNA sequencing analysis and the finding of hemocytes that respond to oxidative stress are important.
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DNA damage signaling in Drosophila macrophages modulates systemic cytokine levels in response 1 to oxidative stress

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By Fabian Hersperger, Tim Meyring, Pia Weber, Chintan Chhatbar
| BioRxiv
Abstract Environmental factors, infection, or injury can cause oxidative stress in diverse tissues and loss of tissue homeostasis. Effective stress response cascades, conserved from invertebrates to mammals, ensure reestablishment of homeostasis and tissue repair. Hemocytes, the Drosophila blood-like cells, rapidly respond to oxidative stress by immune activation.
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Drosophila macrophages control systemic cytokine levels in response to oxidative stress via a non-canonical DNA damage repair signaling cascade

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By Chintan Chhatbar, Fabian Hersperger, Tim Meyring, Pia Weber
| BioRxiv
Thank you for your interest in spreading the word about bioRxiv. NOTE: Your email address is requested solely to identify you as the sender of this article. Your Email * Your Name * Send To * Enter multiple addresses on separate lines or separate them with commas. Message Subject (Your Name) has forwarded a page to you from bioRxiv Message Body (Your Name) thought you would like to see this page from the bioRxiv website.
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