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Valid Gahramanov

As seen in: Nature, MDPI, BioRxiv
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Articles

Stress-coping phenotypes shape adipose tissue function and plasticity and modulate metabolic resilience

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By Saumya Mehta, Michaella Ben-Shachar, Sharmila Govindaraj, Suparna Sen, Oshrit Rahimi, Valid Gahramanov, Galia Luboshits, Michael Firer, Ayala Wollman, Albert Pinhasov, Tovit Rosenzweig, Adrian Segev
| Nature Verified
Abstract Our previous findings demonstrated that stress resilience confers metabolic protection in high-fat diet (HFD)-fed mice. Recognizing the central role of adipose tissue in the pathogenesis of obesity comorbidities, this study aimed to define how stress resilience shapes adipose tissue function. Stress-resilient (Dominant, Dom) and stress-vulnerable (Submissive, Sub) mice were examined under standard (STD) or HFD.
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IJMS | Free Full-Text | Evolution of Resistance to Irinotecan in Cancer Cells Involves Generation of Topoisomerase-Guided Mutations in Non-Coding Genome That Reduce the Chances of DNA Breaks

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By Santosh Kumar, Valid Gahramanov, Shivani Patel, Julia A. Yaglom
| MDPI
This is an early access version, the complete PDF, HTML, and XML versions will be available soon. Open AccessArticle by Santosh Kumar 1, Valid Gahramanov 1, Shivani Patel 1, Julia Yaglom 1, Lukasz Kaczmarczyk 1, Ivan A. Alexandrov 2, Gabi Gerlitz 1, Mali Salmon-Divon 3 and Michael Y.
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Evolution of resistance to Irinotecan in cancer cells involves generation of topoisomerase-guided mutations in non-coding genome that reduce the chances of DNA breaks

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By Santosh Kumar, Shivani Patel, Julia A. Yaglom, Valid Gahramanov
| BioRxiv
Abstract Resistance to chemotherapy is a leading cause of treatment failure. Drug-resistance mechanisms involve mutations in specific proteins or changes in their expression levels. It is commonly understood that resistance mutations happen randomly prior to treatment and are selected during the treatment.
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