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1 Introduction Extracellular matrix (ECM) consisting of noncellular components of the tumor microenvironment (TME) accounts for up to 60% of tumor mass (Henke et al. 2019; Anderson and Simon 2020). This ECM is composed of various fibrous proteins including fibrillar collagens, fibronectin 1 (FN1), and fibulin 2 (FBLN2) (Brassart-Pasco et al. 2020; Cox 2021).
1 INTRODUCTION Coral reefs are biodiversity hotspots in the ocean that nurture approximately one-third of known marine organisms (Fisher et al., 2015). Globally, coral reefs are increasingly under severe stress due to anthropogenic impacts, diseases and predator damage (Hoegh-Guldberg, 1999; Hughes et al., 2017, 2018), with long-term data showing a 50% decrease of global coral coverage since the 1950s (Eddy et al., 2021).
Adult zebrafish are able to heal large-sized cutaneous wounds in hours with little to no scarring. This rapid re-epithelialization is crucial for preventing infection and jumpstarting the subsequent regeneration of damaged tissues. Despite significant progress in understanding this process, it remains unclear how vast numbers of epithelial cells are orchestrated on an organismic scale to ensure the timely closure of millimeter-sized wounds.
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