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Woramate Bhorntarakcharoen on Muck Rack

Woramate Bhorntarakcharoen

As seen in: Nature, Wiley Online Library
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Articles

Efficacy and safety of photothermal biomodulated platelet-rich plasma versus standard platelet-rich plasma for facial rejuvenation: a prospective split-face randomized study

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By Rungsima Wanitphakdeedecha, Woramate Bhorntarakcharoen, Thanya Techapichetvanich, Pitchaya Maneeprasopchoke, Panwadee Thongjaroensirikul, Ya-Nin Nokdhes, Sariya Sittiwanaruk, Wilailuck Phokai, Thanakorn Woramongkol, Pornsuk Yamlexnoi, Onjira Meethong
| Nature Verified
Abstract Autologous platelet-rich plasma (PRP) regenerative properties are used to treat clinical manifestations of skin aging. This study assessed the efficacy and safety of photothermal biomodulated PRP (PTBM-PRP) compared to standard PRP for skin rejuvenation. Prospective, split-face, randomized study including Thai volunteers (30–60 years) with Fitzpatrick skin type III-IV and moderate severity photoaging or higher conducted at Siriraj Hospital (Bangkok, Thailand).
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Prospective Evaluation of a Fractional 675‐nm Nonablative Laser for Atrophic Acne Scars in Fitzpatrick Skin Types III-IV

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By Woramate Bhorntarakcharoen, Pinthisa Nisagornsen, Panwadee Thongjaroensirikul, Yanin Nokdhes
| Wiley Online Library Verified
1 Introduction Acne vulgaris is among the most common dermatologic conditions, affecting up to 85% of adolescents and young adults worldwide, with persistence into adulthood in approximately 9.4% of individuals [1, 2]. Although often self-limiting, acne frequently leads to atrophic scarring due to inflammatory disruption of follicular and dermal structures.
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One‐Year Follow‐Up of Acne Vulgaris Patients Treated With Nonablative Monopolar Radiofrequency: Efficacy and Safety Assessment

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By Woraphong Manuskiatti, Supisara Wongdama, Jayne Li, Woramate Bhorntarakcharoen
| Wiley Online Library Verified
1 Introduction Acne vulgaris (AV) is a chronic inflammatory condition of the pilosebaceous unit that affects a substantial percentage of individuals worldwide [1, 2]. Its multifactorial pathogenesis involves a combination of intrinsic and extrinsic factors. Key mechanisms include excessive sebum production, follicular hyperkeratinization, inflammation, and colonization by Cutibacterium acnes [2, 3].
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