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Abstract Liver fibrosis represents a major global health burden, with its core pathological feature being the excessive deposition of extracellular matrix driven by activated hepatic stellate cells (HSCs). Acid-sensitive ion channel 1a (ASIC1a) has been shown to be associated with the progression of liver fibrosis, but the molecular mechanisms by which it regulates RNA m6A methylation remain unclear.
Abstract Liver diseases are hepatic pathological changes caused by a variety of pathogenic factors, including viral hepatitis, alcoholic liver disease (ALD), metabolic dysfunction-associated steatotic liver disease (MASLD), cirrhosis, and hepatocellular carcinoma (HCC), etc. The characteristics of liver diseases are steatosis, hepatocyte ballooning, and immune cell infiltration. Among these immune cells, neutrophils are important immune cells in innate immunity.
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