Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile
Highlights • Mice with 500 C9ORF72 repeats expressed repeat-containing RNA and dipeptide proteins • In two distinct labs, no survival or motor deficits were found linked to the transgene • Neurological phenotypes inherent to FVB mice complicate their use in ALS/FTD research Summary , published in A hexanucleotide repeat expansion at C9ORF72 is the most common genetic cause of amyotrophic lateral sclerosis (ALS)/frontotemporal dementia (FTD).
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
