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Submitted: 05 October 2026 You are already at the latest version Neurodegenerative diseases (NDs) have traditionally been regarded as brain-centered disorders driven primarily by the accumulation of pathological proteins. However, increasing evidence indicates that systemic metabolic dysfunction, chronic inflammation, intestinal dysbiosis, hepatic abnormalities, and barrier impairment may substantially influence disease susceptibility and progression.
You are already at the latest version Alzheimer’s Disease (AD) is now understood as a biologically diverse condition, with amyloid and tau pathology evolving within dynamic neuroimmune networks. This challenges the traditional view that AD-related inflammation can be broadly suppressed therapeutically. We review evidence showing that neuroinflammation in AD is stage-dependent, cell-state-specific, spatially organized, and functionally complex.
arXiv:2401.12246 (cs) Download PDF HTML (experimental) Comments: Subjects: Computation and Language (cs.CL); Machine Learning (cs.LG) Cite as: arXiv:2401.12246 [cs.CL] (or arXiv:2401.12246v1 [cs.CL] for this version) https://doi.org/10.48550/arXiv.2401.12246 Submission history From: Haihui Pan [ view email] [v1] Sat, 20 Jan 2024 12:29:27 UTC (525 KB) Bibliographic Tools Bibliographic Explorer Toggle Bibliographic Explorer () Litmaps Toggle Litmaps (What is Litmaps?) scite.ai Toggle scite...
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