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Articles by Xiaoying Qiao
Ketogenic diet in treating NADH accumulation caused by complex I dysfunction: the metabolic wisdom of “dredging, not repairing”
Abstract Genetic mutations causing Complex I (CI) dysfunction lead to the accumulation of NADH in the mitochondrial matrix due to impaired oxidation. An elevated NADH/NAD⁺ ratio feedback-inhibits key enzymes of the tricarboxylic acid (TCA) cycle, reducing metabolic flux, while concurrent electron transport chain (ETC) blockade exacerbates reactive oxygen species (ROS) generation and oxidative stress.
Efficient Data Sampling and Feature Selection Algorithms for Scalable Machine Learning Pipelines
You are already at the latest version As machine learning pipelines grow to handle massive datasets, the computational cost of data processing and feature engineering becomes a primary bottleneck. Traditional pipelines often process all available data uniformly, leading to inefficiencies when large portions of the data contribute little to model improvement.
Loss of Bone Morphogenetic Protein-9 Reduces Survival and Increases MMP Activity After Myocardial Infarction:
Introduction Myocardial infarction (MI) is a major cause of global morbidity and mortality that affects nearly 800,000 patients each year in the United States alone.1 Cardiac wound healing after MI is a complex and multiphase process that begins with necrosis and apoptosis of cardiomyocytes due to ischemic injury with or without reperfusion followed by the formation of a fibrotic scar.2 The acute phase of this process involves an inflammatory response and degradation of the extracellular...
Transvalvular Unloading Mitigates Ventricular Injury Due to Venoarterial Extracorporeal Membrane Oxygenation in Acute Myocardial Infarction:
JACC Journals › JACC: BTS › Archives › Vol. 8 No. 7 Highlights • Transvalvular unloading with VA-ECMO (EC-Pella) is being used in patients with AMI. • VA-ECMO increases IS despite reducing LV work while EC-Pella reduces IS and LV work. • Cardioprotective signaling is activated with EC-Pella, not VA-ECMO. • EC-Pella fails to rescue mitochondrial injury due to VA-ECMO. • Load independent myocardial injury mechanisms due to VA-ECMO may be targets in AMI.
Transvalvular Ventricular Unloading Before Reperfusion in Acute Myocardial Infarction
Journal of the American College of Cardiology Original Investigation Lija Swain, Lara Reyelt, Shreyas Bhave, Xiaoying Qiao, Corinne J. Thomas, Elric Zweck, Paige Crowley, Courtney Boggins, Michele Esposito, Michael Chin, Richard H. Karas, William O’Neill and Navin K. Kapur This article requires a subscription or purchase to view the full text. If you are a subscriber or member, click the login link or the subscribe link in the top menu above to access this article.
Reduced Activin Receptor Like-Kinase 1 Activity Promotes Cardiac Fibrosis in Heart Failure
Publication HistoryPublished online: July 17, 2017Accepted: July 12, 2017; Received in revised form: July 3, 2017; Received: May 13, 2017;
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