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Xingying Zeng on Muck Rack

Xingying Zeng

As seen in: Frontiers, Frontiers in Immunology, MDPI
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Articles

Clinical application of HMGB1-TLR4 signaling pathway-mediated neuroinflammatory markers in infantile epileptic spasms syndrome

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By Hui Chen, Jianmin Zhong, Yong Chen, Huaping Wu, Ruiyan Wang, Zhaoshi Yi, Xingying Zeng
| Frontiers in Immunology
Abstract Background: Infantile epileptic spasms syndrome (IESS) is a severe age-specific epileptic encephalopathy with unclear pathogenesis, and neuroinflammation is involved in its progression. The HMGB1-TLR4 signaling pathway, a key neuroinflammatory mediator in various epilepsies, has not been studied for its role and clinical biomarker potential in IESS. Methods: A retrospective study included 66 IESS patients treated with a modified prednisone regimen and 53 age-matched healthy controls.
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HMGB1-TLR4 signaling-mediated neuroinflammation contributes to the pathogenesis of infantile epileptic spasms syndrome in rats

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By Hui Chen, Jianmin Zhong, Yong Chen, Huaping Wu, Ruiyan Wang, Zhaoshi Yi, Xingying Zeng
| Frontiers
Abstract Background: Infantile epileptic spasm syndrome (IESS) is a severe age-dependent epileptic encephalopathy in infancy with poor prognosis and unclear pathogenesis. Neuroinflammation plays a pivotal role in epileptogenesis, and the high-mobility group box 1 protein (HMGB1)-Toll-like receptor 4 (TLR4) axis acts as a core mediator of neuroinflammation. However, its specific role in IESS remains elusive.
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RNA-Seq Analysis Uncovered Transcriptomic Changes in Poncirus trifoliata Roots Under Long-Term Soil Drought Conditions

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By Lin Zheng, qin huang, Chuncao Song, Xingying Zeng
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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