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Yadi Huang

As seen in: Frontiers, MDPI, BioRxiv
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Articles

Age- and diet-dependent progression of retinal microvascular injury in GCK-MODY under metabolic stress

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By Yadi Huang, Yang Liu, Yiqing Wang, Xuan Liu, Shuhui Ji, Shanshan Chen, Hua Shu, yu xin fan, Ming Liu Verified, Xin Li
| Frontiers
Abstract Background: Maturity-onset diabetes of the young type 2 (GCK-MODY), caused by heterozygous inactivating mutations in the glucokinase (GCK) gene, is generally considered a mild and stable form of diabetes with a relatively low risk of chronic complications. However, whether GCK deficiency predisposes to retinal microvascular injury under metabolic stress remains unclear.
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Distinct duration and diet dependent lipid profiles and renal complicationsin GCK-MODY

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By Yadi Huang, Yuxin Fan, Yang Liu, Xuan Liu
| BioRxiv
Abstract Heterozygous inactivating mutations in the glucokinase (GCK) gene cause one of the most common types of maturity-onset diabetes of the young type 2 (MODY2), also named GCK-MODY. Studies suggest that, unlike other types of diabetes, patients with GCK-MODY do not have increased risk of diabetic complications and therefore do not typically require antihyperglycemic therapy. However, long-term outcomes of GCK-MODY on lipid profiles and chronic complications remain unclear.
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