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Yete G. Ferri

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Articles

Reduced IP6K levels impair life-cycle transitions and intracellular development of Trypanosoma cruzi within human cardiomyocytes

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By Thaise L. Teixeira, Vitor da Silva, Anuj K Shukla, Carolina M. C. Catta-Preta, Craig Lapsley, Samuel Teixeira, Miguel A. Chiurillo, Noelia Lander, Eduardo Reis, Henning J. Jessen, Richard McCulloch, Marcelo da Silva, Bryan E. Abuchery, Rauni B. Marques, Fernanda M. Gerolamo, Bruno A. Santarossa, Yete G. Ferri, Maria Cristina M. Motta, Simone G. Calderano
| Nature Verified
Abstract Trypanosoma cruzi is the etiological agent of Chagas disease (CD). During its life cycle, T. cruzi undergoes several key differentiation processes that are essential for its survival. The precise mechanisms that regulate these processes remain elusive, and any interference in this cycle would represent a breakthrough in the development of effective therapy against CD. Here we show that key differentiation processes of T.
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