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Ying-Lien Chen

As seen in: Nature, American Society for Microbiology (ASM), Journal of Infection
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Articles

Rad6 and Bre1 ubiquitin ligase negatively regulate biofilm formation and virulence in Candida glabrata

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By Yi-Hang Lee, Li-Hang Hsu, Zi-Heng Yu, Kai-Quan Leong, Hao-Sen Chiang, Ying-Lien Chen
| Journal of Infection Verified
Keywords Candida glabrata Rad6 Bre1 biofilm formation drug resistance oxidative stress virulence macrophage Introduction Invasive fungal infections have emerged as a critical global health concern, with Candida species being major opportunistic pathogens causing significant morbidity and mortality in immunocompromised patients.1,2 Among these species, C.
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Author Correction: Harnessing calcineurin-FK506-FKBP12 crystal structures from invasive fungal pathogens to develop antifungal agents - Nature Communications

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By Joshua D. Wheaton, Maria Ciofani, soo lee, Ying-Lien Chen
| Nature Verified
Correction to: Nature Communications https://doi.org/10.1038/s41467-019-12199-1, published online 19 September 2019 In the version of the article initially published, in Fig. 4a, the first and second images in row “K53T-Q54G” were inadvertently duplicated from the corresponding images in row “R14V-K45E”. The corrected Fig. 4 is available as Fig. 1, below. This notice serves to amend the error. Fig. 1 | Corrected Fig.
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Deletion of ADA2 Increases Antifungal Drug Susceptibility and Virulence in Candida glabrata

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By Shang-Jie Yu, Ya-Lin Changa, Ya-Lin Chang, Ying-Lien Chen
| American Society for Microbiology (ASM)
Candida glabrata, the second most frequent cause of candidiasis after Candida albicans, is an emerging human fungal pathogen that is intrinsically drug tolerant. Currently, studies of C. glabrata genes involved in drug tolerance are limited. Ada2, a component serving as a transcription adaptor of the Spt-Ada-Gcn5 acetyltransferase (SAGA) complex, is required for antifungal drug tolerance and virulence in C. albicans. However, its roles in C. glabrata remain elusive.
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