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Ying-Ling Chen on Muck Rack

Ying-Ling Chen

As seen in: Nature, MDPI
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Articles

PARK19 truncation mutant Dnajc6 causes lysosomal deficiency-induced upregulation of pathologic α-synuclein and neurodegeneration of substantia nigra dopaminergic cells in PARK19 knockin mice

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By Ying-Ling Chen, Ching-Chi Chiu, Yi-Hsin Weng, Shu-Yu Liu, Allen Li, Tu-Hsueh Yeh, Hung-Li Wang, Tai-Ju Chiu
| Nature Verified
Abstract Homozygous (Q789X) DNAJC6 mutation causes PARK19. Q787 of Dnajc6 corresponds to Q789 of DNAJC6. Dnajc6Q787X/Q787X mouse was utilized to elucidate pathomechanisms underlying (Q789X) DNAJC6-induced PARK19. Dnajc6Q787X/Q787X mice displayed PARK19 motor deficits and degeneration of substantia nigra (SN) dopaminergic neurons.
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IJMS | Free Full-Text | Downregulation of Protease Cathepsin D and Upregulation of Pathologic α-Synuclein Mediate Paucity of DNAJC6-Induced Degeneration of Dopaminergic Neurons

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By Ching-Chi Chiu, Ying-Ling Chen, Yi-Hsin Weng, Shu-Yu Liu
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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