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Ying Shen on Muck Rack

Ying Shen

As seen in: Nature, Wiley Online Library, arXiv, PLOS, BMJ Open, Cisco Blogs, Frontiers, Frontiers in Medicine, Frontiers in Immunology, Frontiers in Public Health and
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Articles

IRF1-mediated thioredoxin (TXN) protects multiple myeloma cells from ferroptosis through remodeling of PUFA-containing phospholipids

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By Jiaxi Liu, Jiyu Miao, Yang Lv, Bingjie Fu, Xiaomin Ren, Naiqi Wang, Wenxiang Zhang, Bide Zhao, Ying Shen, Wanhong Zhao, Rui Liu, Aili He, Xuezhu Xv, Gongzhizi Gao, Zujie Lin, Yachun Jia
| Nature Verified
Abstract Multiple myeloma (MM) is a hematologic malignancy characterized by uncontrolled plasma cell proliferation and frequent therapeutic relapse. Ferroptosis, an iron-dependent form of regulated cell death driven by lipid peroxidation, has emerged as a promising therapeutic vulnerability in cancer. However, the mechanisms governing ferroptosis resistance in MM remain incompletely understood.
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Comparison of extracorporeal shock wave therapy targeting the posterior myofascial chain and plantar tenderness points versus plantar tenderness points alone in patients with plantar fasciitis: a randomized controlled trial

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By Shichong Wang, Baoshan Xu, Chuan Guo, Ying Shen, Siyifan Chen, Gaojian Tao
| Frontiers in Medicine
Abstract Aim: Plantar fasciitis is a common cause of heel pain. Although extracorporeal shock wave therapy (ESWT) targeting plantar tenderness points is widely used, outcomes remain unsatisfactory in some patients. This study aimed to compare the clinical efficacy of ESWT targeting both posterior myofascial-chain trigger points and plantar tenderness points with that of conventional ESWT targeting plantar tenderness points alone in patients with plantar fasciitis.
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Integrated analysis of single-cell and transcriptome data reveals temporal dynamics of microglial phagocytic reprogramming in neuropathic pain progression

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By Yilun Qian, Yu Wang, Yan Liu, Yao Geng, Manyu Dong, Ying Huang, Chenyuan Zhai, Qinfeng Wu, Wentao Liu, Ying Shen, Qi Wu, Shangbo Niu
| Nature Verified
Abstract Neuropathic pain (NP) often progresses from acute to chronic, but the mechanisms driving this transition remain unclear. Microglia play a central role in spinal sensitization, yet their functional evolution during pain chronification is poorly understood. Through integrated multi-omics analysis and experimental validation, this study systematically investigates the temporal dynamics of microglial phagocytic reprogramming during the progression of neuropathic pain (NP).
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