Yinghua Jiang
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Itaconate restrains acute proinflammatory activation of microglia MG after traumatic brain injury in mice
Editor’s summary Microglia can have beneficial or detrimental effects on recovery after traumatic brain injury (TBI), but it is incompletely understood which factors determine microglial phenotype transitions.
Tracing the path of disruption: 13C isotope applications in traumatic brain injury‐induced metabolic dysfunction
1 INTRODUCTION Traumatic brain injury (TBI) remains a significant public health challenge with limited therapeutic options.1 Developing new treatments based on mechanistic rationale is crucial for improving outcomes across diverse TBI patients.
Advancement of epigenetics in stroke
Introduction Stroke is one of the main leading causes of death and the first leading cause of disability worldwide (Avan et al., 2019; Collaborators, 2019). Hemorrhagic stroke, including intracerebral hemorrhage (ICH) and subarachnoid hemorrhage (SAH), happens when a blood vessel in the brain bursts or when brain tissue starts to bleed.
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