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Publisher Correction: Rates, distribution and implications of postzygotic mosaic mutations in autism spectrum disorder
Correction to: Nature Neuroscience https://doi.org/10.1038/nn.4598, published online 17 July 2017. In the version of this article initially published, in Fig. 2a, zeros were omitted from the y-axis labels on the histogram. They should read, from bottom to top: 0.01, 0.02, 0.03, 0.04, 0.05, 0.06, 0.07. The error has not been corrected in the original article.
An enhanced CRISPR repressor for targeted mammalian gene regulation
Y.C., D. Menn, D. Milanova, and C.-C.K. contributed equally to the manuscript. A.C. and N.C.Y. conceived the study. N.C.Y. and A.C. designed experiments. A.C. and A.T. designed, built, and tested the initial set of dCas9 repressor fusions. N.C.Y. performed the majority of endogenous gene targeting experiments with assistance from A.L.-B.
High-throughput creation and functional profiling of DNA sequence variant libraries using CRISPR-Cas9 in yeast
Mullen, J.R., Kaliraman, V., Ibrahim, S.S. & Brill, S.J. Requirement for three novel protein complexes in the absence of the Sgs1 DNA helicase in Saccharomyces cerevisiae . Genetics 157, 103–118 (2001). Ui, A. et al. The ability of Sgs1 to interact with DNA topoisomerase III is essential for damage-induced recombination . DNA Repair (Amst.) 4, 191–201 (2005).
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