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Yingli Gu

As seen in: Nature, Genome Research
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Articles

Single-nucleus multiomic profiling of the aging mouse substantia nigra reveals conserved gene alterations linked to Parkinson's disease

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By Kangli Wang, Weikun Xia, Yingli Gu
| Genome Research
1,2,5,6 1Department of Cellular and Molecular Medicine, University of California San Diego, School of Medicine, La Jolla, California 92093, USA; 2Center for Epigenomics, University of California San Diego, School of Medicine, La Jolla, California 92093, USA; 3Westlake Laboratory of Life Sciences and Biomedicine, School of Life Sciences, Westlake University, Hangzhou, Zhejiang 310024, China; 4Department of Neuroscience, University of California San Diego, La Jolla, California 92093, USA...
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Single-nucleus multiomic profiling of the aging mouse substantia nigra reveals conserved gene alterations linked to Parkinson's disease

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By Kangli Wang, Weikun Xia, Yingli Gu
| Genome Research
1,4 1 University of California San Diego, School of Medicine; 2 Westlake University; 3 University of California San Diego ↵* Corresponding author; email: br2833{at}cumc.columbia.edu Received June 23, 2025. Accepted March 2, 2026. This manuscript is Open Access. This article, published in Genome Research, is available under a Creative Commons License (Attribution-NonCommercial 4.0 International license), as described at http://creativecommons.org/licenses/by-nc/4.0/.
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CryoET reveals organelle phenotypes in huntington disease patient iPSC-derived and mouse primary neurons - Nature Communications

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By Charlene Smith-Geater, Patrick Mitchell, Joy Hsu, Lydia-Marie Joubert, Michael Schmid, Serena Yeung, Judith Frydman, Leslie Thompson, Yingli Gu, Keona Q. Wang
| Nature Verified
Abstract Huntington’s disease (HD) is caused by an expanded CAG repeat in the huntingtin gene, yielding a Huntingtin protein with an expanded polyglutamine tract. While experiments with patient-derived induced pluripotent stem cells (iPSCs) can help understand disease, defining pathological biomarkers remains challenging.
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