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Yingqing Wang

As seen in: Nature, MDPI
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Articles

SAK3, a Cav3.1 enhancer facilitates mitochondrial function and neuronal survival in Parkinson’s disease models by modulating the SphK1/S1P signaling cascade

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By Lina Chen, Yingqing Wang, Zhiting Chen, Haoling Xu, Xiaochun Chen, En Huang, Guoen Cai, Yuqi Zeng, Qinyong Ye, Jiaqia Ke
| Nature Verified
Abstract Parkinson’s disease (PD) involves dopaminergic neuron loss linked to calcium imbalance. Although calcium channel blockers have shown limited efficacy, SAK3 is a known selective enhancer of the T-type calcium channel Cav3.1. While it shows neuroprotective potential, its precise downstream mechanisms in Parkinson’s disease (PD) remain unclear. We found Cav3.1, the principal T-type channel in the substantia nigra, is downregulated in PD models and patient tissues.
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Characterization of blood metabolomic responses to deep brain stimulation in patients with Parkinson’s disease

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By Fabin Lin, Lebo Zhou, Lina Chen, Yufeng Xie, Yingqing Wang, Yuqi Zeng, Zhen Chen, Qinyong Ye, Woo-ping Ge, Guoen Cai
| Nature Verified
Abstract Deep brain stimulation (DBS) constitutes one of the few therapeutic strategies for advanced Parkinson’s disease. Although DBS causes rapid alterations in neuronal metabolism, whether it remodels the circulating metabolome and thereby alleviates systemic symptoms remains unknown.
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β Oscillations of Dorsal STN as a Potential Biomarker in Parkinson’s Disease Motor Subtypes: An Exploratory Study

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By Yongjie Li, Yuqi Zeng, Mangui Lin, Yingqing Wang
| MDPI
This is an early access version, the complete PDF, HTML, and XML versions will be available soon.
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