Skip To Main Content
Yingxi Li on Muck Rack

Yingxi Li

As seen in: Nature, Wiley Online Library, Frontiers, MDPI, BMC Cancer, Canadian Journal of Cardiology
Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work. Claim your profile

Get in touch with Yingxi

Contact Yingxi, search articles and posts on X, monitor coverage, and track replies from one place.

Learn more about Muck Rack

Actions

Share this page

Is this you?

As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile

Articles

Marigold flavonoids coordinate emulsion stabilization, epithelial transport and gut microbiota modulation to enhance intestinal lutein absorption

|
By Sitong Wang, Zhiyue Xu, Yingxi Li
| Wiley Online Library Verified
DATA AVAILABILITY STATEMENT The data that support the findings of this study are available from the corresponding author upon reasonable request. REFERENCES 1, , , , , et al., Imaging lutein and zeaxanthin in the human retina with confocal resonance Raman microscopy. Proc Natl Acad Sci 117: 12352–12358 (2020). https://doi.org/10.1073/pnas.1922793117.
Open in Who Shared Issue with article?

Integrative single-cell and genomic analysis reveals NMB as a driver of metastatic adaptation in esophageal squamous cell carcinoma via metabolic rewiring and immune evasion

|
By Long He, Jiahui Zhang, Jing Hui Zeng, Yao Tian, Xiaofeng Liu, Yingxi Li, Zhikai Cao, Dongchen Tian
| Frontiers
Abstract Background: Esophageal squamous cell carcinoma (ESCC) has high mortality, and metastasis is the leading cause of patient death. Neuromedin B (NMB) promotes tumor development in various cancers, yet its role in ESCC metastasis remains unclear. Methods: We integrated single-cell transcriptomic data from matched primary and metastatic ESCC lesions (GSE309392) with bulk transcriptomic cohorts from TCGA and GSE53624.
Open in Who Shared Issue with article?

Necroptosis is a key contributor to impaired cardiac repair following myocardial infarction

|
By Razoan Al Rimon, Yingxi Li, Ilamaran Meganathan, Faqi Wang, Allan Murray, Gavin Y. Oudit, Slava Epelman, Xavier Clemente-Casares, Zamaneh Kassiri
| Nature Verified
Abstract Myocardial infarction (MI) is a leading cause of morbidity and death worldwide. Endothelial cells (ECs) contribute to post-MI remodeling through angiogenesis, inflammation, and endothelial-to-mesenchymal transition (EndMT). ADAM17, a membrane-bound protease, is upregulated in ischemic heart disease, but its role in endothelial function post-MI is unknown.
Open in Who Shared Issue with article?
See all 11 articles